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Table 2 Findings in microscopic analysis of MIA tissue samples

From: Assessing viability of a minimally invasive autopsy technique in ascertaining the probable cause of death in patients who were SARS CoV19 positive at the time of their demise

Histopathological finding

N (%)

Lung (49)

 Diffuse alveolar damage (DAD)

44 (89.79)

  Early DAD

14 (28.57)

  Proliferative or organising phase of DAD

20 (40.81)

  Late DAD

10 (20.40)

 Interstitial pneumonia

4 (8.16)

 Lung abscess

1 (2.04)

 Pulmonary oedema

6 (12.24)

 Microthrombi

21 (42.85)

 Hyaline membrane

19 (39.77)

 Type II pneumocyte hyperplasia

15 (30.61)

 Intra-alveolar hemorrhage

12 (24.48)

Myocardium (51)

 Focal myocardial hypertrophy

39 (79.59)

 Interstitial myocarditis

2 (4.08)

 Interstitial fibrosis

8 (16.32)

 Plasma cell and neutrophilic infiltration

6 (12.24)

Kidney (47)

 Cloudy degeneration of tubules

24 (51.06)

 Thyroidisation of tubules

7 (14.89)

 Tubular atrophy

6 (12.76)

 Glomerular congestion

18 (38.29)

 Mesangial proliferation

12 (25.53)

 Glomerulosclerosis

7 (14.89)

 Interstitial inflammation

12 (25.53)

 Hyaline arteriosclerosis

9 (19.14)

Liver (49)

 Hepatic sinusoidal dilatation and congestion

29 (59.18)

 Vacuolar degeneration

22 (44.89)

 Microvesicular fatty change

18 (36.3)

 Cholestasis

5 (10.20)

 Lymphocytic infiltration

30 (61.22)

 Central venous congestion

14 (28.57)

Spleen (46)

 Sinusoidal congestion

22 (47.83)

 Amyloidosis

1 (2.17)