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Table 2 Findings in microscopic analysis of MIA tissue samples

From: Assessing viability of a minimally invasive autopsy technique in ascertaining the probable cause of death in patients who were SARS CoV19 positive at the time of their demise

Histopathological finding N (%)
Lung (49)
 Diffuse alveolar damage (DAD) 44 (89.79)
  Early DAD 14 (28.57)
  Proliferative or organising phase of DAD 20 (40.81)
  Late DAD 10 (20.40)
 Interstitial pneumonia 4 (8.16)
 Lung abscess 1 (2.04)
 Pulmonary oedema 6 (12.24)
 Microthrombi 21 (42.85)
 Hyaline membrane 19 (39.77)
 Type II pneumocyte hyperplasia 15 (30.61)
 Intra-alveolar hemorrhage 12 (24.48)
Myocardium (51)
 Focal myocardial hypertrophy 39 (79.59)
 Interstitial myocarditis 2 (4.08)
 Interstitial fibrosis 8 (16.32)
 Plasma cell and neutrophilic infiltration 6 (12.24)
Kidney (47)
 Cloudy degeneration of tubules 24 (51.06)
 Thyroidisation of tubules 7 (14.89)
 Tubular atrophy 6 (12.76)
 Glomerular congestion 18 (38.29)
 Mesangial proliferation 12 (25.53)
 Glomerulosclerosis 7 (14.89)
 Interstitial inflammation 12 (25.53)
 Hyaline arteriosclerosis 9 (19.14)
Liver (49)
 Hepatic sinusoidal dilatation and congestion 29 (59.18)
 Vacuolar degeneration 22 (44.89)
 Microvesicular fatty change 18 (36.3)
 Cholestasis 5 (10.20)
 Lymphocytic infiltration 30 (61.22)
 Central venous congestion 14 (28.57)
Spleen (46)
 Sinusoidal congestion 22 (47.83)
 Amyloidosis 1 (2.17)